Endothelial nitric oxide synthase limits left ventricular remodeling after myocardial infarction in mice

Circulation

Volumn 104, Issue 11, 2001, Pages 1286-1291

  Scherrer Crosbie, Marielle ^a   Ullrich, Roman ^a   Bloch, Kenneth D ^a   Nakajima, Hiroshi ^a   Nasseri, Boris ^a   Aretz, H Thomas ^a   Lindsey, Merry L ^b   Vançon, Anne Claire ^a   Huang, Paul L ^a   Lee, Richard T ^b   Zapol, Warren M ^a   Picard, Michael H ^a  

^a MASSACHUSETTS GENERAL HOSPITAL   (United States)

^b HARVARD MEDICAL SCHOOL   (United States)

Author keywords

Echocardiography; Hypertrophy; Ventricles

Indexed keywords

HYDRALAZINE; NITRIC OXIDE SYNTHASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLOOD PRESSURE REGULATION; CAPILLARY DENSITY; CONTROLLED STUDY; ECHOCARDIOGRAPHY; HEART AFTERLOAD; HEART INFARCTION; HEART LEFT VENTRICLE EJECTION FRACTION; HEART LEFT VENTRICLE ENDDIASTOLIC VOLUME; HEART LEFT VENTRICLE FUNCTION; HEART LEFT VENTRICLE MASS; HEART LEFT VENTRICLE PRESSURE; HEART LEFT VENTRICLE RELAXATION; HEART LEFT VENTRICLE SIZE; HEART MUSCLE CELL; HEART VENTRICLE CONTRACTION; HEART VENTRICLE HYPERTROPHY; HEART VENTRICLE REMODELING; MORTALITY; MOUSE; NONHUMAN; PRIORITY JOURNAL; VASCULAR ENDOTHELIUM;

EID: 0035845630     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/hc3601.094298     Document Type: Article

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