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Volumn 89, Issue 7, 2004, Pages 868-869

TP53 codon 72 polymorphism in patients with chronic myeloid leukemia

Author keywords

Chronic myeloid leukemia; Imatinib mesylate; Polymorphism; TP53

Indexed keywords

IMATINIB;

EID: 3242803705     PISSN: 03906078     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article
Times cited : (40)

References (10)
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  • 3
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    • High frequency of point mutations clustered within the adenosine triphosphate-binding region of BCR/ABL in patients with chronic myeloid leukemia or Ph-positive acute lymphoblastic leukemia who develop imatinib (STI571) resistance
    • Branford S, Rudzki Z, Walsh S, Grigg A, Arthur C, Taylor K, et al. High frequency of point mutations clustered within the adenosine triphosphate-binding region of BCR/ABL in patients with chronic myeloid leukemia or Ph-positive acute lymphoblastic leukemia who develop imatinib (STI571) resistance. Blood 2002;99:3472-5.
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  • 4
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    • Marktel S, Marin D, Foot N, Szydlo R, Bua M, Karadimitris A, et al. Chronic myeloid leukemia in chronic phase responding to imatinib: the occurrence of additional cytogenetic abnormalities predicts disease progression. Haematologica 2003;88:260-7.
    • (2003) Haematologica , vol.88 , pp. 260-267
    • Marktel, S.1    Marin, D.2    Foot, N.3    Szydlo, R.4    Bua, M.5    Karadimitris, A.6
  • 8
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    • O'Dwyer ME, Mauro MJ, Blasdel C, Farnsworth M, Kurilik G, Hsieh YC, et al. Clonal evolution and lack of cytogenetic response are adverse prognostic factors for hematologic relapse of chronic phase CML patients treated with imatinib mesylate. Blood 2004;103:451-5.
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  • 9
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.