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Volumn 99, Issue 9, 2002, Pages 3472-3475

High frequency of point mutations clustered within the adenosine triphosphate-binding region of BCR/ABL in patients with chronic myeloid leukemia or Ph-positive acute lymphoblastic leukemia who develop imatinib (STI571) resistance

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATE; BCR ABL PROTEIN; GLIVIC; IMATINIB;

EID: 0036566540     PISSN: 00064971     EISSN: None     Source Type: Journal    
DOI: 10.1182/blood.V99.9.3472     Document Type: Article
Times cited : (587)

References (19)
  • 3
    • 0035810142 scopus 로고    scopus 로고
    • Activity of a specific inhibitor of the BCR-ABL tyrosine kinase in the blast crisis of chronic myeloid leukemia and acute lymphoblastic leukemia with the Philadelphia chromosome
    • (2001) N Engl J Med , vol.344 , pp. 1038-1042
    • Druker, B.J.1    Sawyers, C.L.2    Kantarjian, H.3
  • 7
    • 0034210577 scopus 로고    scopus 로고
    • Mechanism of resistance to the ABL tyrosine kinase inhibitor sTI571 in BCR/ABL-transformed hematopoietic cell lines
    • (2000) Blood , vol.95 , pp. 3498-3505
    • Weisberg, E.1    Griffin, J.D.2
  • 12
    • 0034093924 scopus 로고    scopus 로고
    • A novel BCR-ABL transcript (e8a2) with the insertion of an inverted sequence of ABL intron 1 b in a patient with Philadelphia-positive chronic myeloid leukaemia
    • (2000) Br J Haematol , vol.109 , pp. 635-637
    • Branford, S.1    Rudzki, Z.2    Hughes, T.P.3
  • 15
    • 0034634597 scopus 로고    scopus 로고
    • c-Abl has high intrinsic tyrosine kinase activity that is stimulated by mutation of the Src homology 3 domain autophosphorylation at two distinct regulatory tyrosines
    • (2000) J Biol Chem , vol.275 , pp. 35631-35637
    • Brasher, B.B.1    Van Etten, R.A.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.