Selenium reduces the proapoptotic signaling associated to NF-κB pathway and stimulates glutathione peroxidase activity during excitotoxic damage produced by quinolinate in rat corpus striatum

Synapse

Volumn 58, Issue 4, 2005, Pages 258-266

  Santamaría, Abel ^a,b   Vázquez Román, Beatriz ^a   Pérez De La Cruz, Verónica ^a   González Cortés, Carolina ^a   Trejo Solís, Ma Cristina ^a   Galván Arzate, Sonia ^a   Jara Prado, Aurelio ^a   Guevara Fonseca, Jorge ^a   Ali, Syed F ^b  

^a NATIONAL INSTITUTE OF NEUROLOGY AND NEUROSURGERY   (Mexico)

^b NATIONAL CENTER FOR TOXICOLOGICAL RESEARCH   (United States)

Author keywords

Antioxidant defense; Excitotoxicity; NF B pathway; NMDA receptor

Indexed keywords

CASPASE 3; DNA FRAGMENT; GLUTATHIONE PEROXIDASE; I KAPPA B ALPHA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; QUINOLINIC ACID; SELENIUM; SODIUM SELENITE; SYNAPTOTAGMIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRAIN INJURY; CELL DAMAGE; CONTROLLED STUDY; CORPUS STRIATUM; ENZYME ACTIVATION; ENZYME ACTIVITY; EXCITOTOXICITY; IMMUNOCOMPETENT CELL; IMMUNOHISTOCHEMISTRY; MALE; NEUROTOXICITY; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION; RAT; SIGNAL TRANSDUCTION;

ANIMALS; BLOTTING, WESTERN; CORPUS STRIATUM; DNA FRAGMENTATION; GLUTATHIONE PEROXIDASE; I-KAPPA B PROTEINS; IMMUNOHISTOCHEMISTRY; MALE; NEUROPROTECTIVE AGENTS; NF-KAPPA B; QUINOLINIC ACID; RATS; RATS, WISTAR; SELENIUM; SIGNAL TRANSDUCTION;

EID: 28044431816     PISSN: 08874476     EISSN: None     Source Type: Journal    
DOI: 10.1002/syn.20206     Document Type: Article

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