Cardiac-specific attenuation of natriuretic peptide A receptor activity accentuates adverse cardiac remodeling and mortality in response to pressure overload

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 289, Issue 2 58-2, 2005, Pages

  Patel, Jeetendra B ^a   Valencik, Maria L ^b   Pritchett, Allison M ^a,c   Burnett Jr , John C ^a   McDonald, John A ^b   Redfield, Margaret M ^a  

^a MAYO CLINIC   (United States)

^b UNIVERSITY OF NEVADA SCHOOL OF MEDICINE   (United States)

^c BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

Diastole; Fibrosis; Hypertrophy; Natriuretic peptides

Indexed keywords

ATRIAL NATRIURETIC FACTOR; ATRIAL NATRIURETIC FACTOR RECEPTOR; BRAIN NATRIURETIC PEPTIDE; CYCLIC GMP;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOCRINE EFFECT; BLOOD PRESSURE REGULATION; CONTROLLED STUDY; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; HEART VENTRICLE OVERLOAD; HEART VENTRICLE PRESSURE; HEART VENTRICLE REMODELING; HISTOPATHOLOGY; HORMONE RECEPTOR INTERACTION; MALE; MORTALITY; MOUSE; NONHUMAN; PARACRINE SIGNALING; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ANIMALS; AORTA; ATRIAL NATRIURETIC FACTOR; BLOOD PRESSURE; CYCLIC GMP; ECHOCARDIOGRAPHY; GENE EXPRESSION; GENES, DOMINANT; GUANYLATE CYCLASE; HYPERTENSION; LIGATION; MICE; MICE, INBRED STRAINS; MICE, TRANSGENIC; MUTATION; MYOCARDIUM; RECEPTORS, ATRIAL NATRIURETIC FACTOR; TRANSGENES; VENTRICULAR REMODELING;

EID: 23744439052     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00117.2005     Document Type: Article

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