The guanylyl cyclase-deficient mouse defines differential pathways of natriuretic peptide signaling

Journal of Biological Chemistry

Volumn 272, Issue 37, 1997, Pages 23064-23068

  Lopez, M James ^a   Garbers, David L ^a,b   Kuhn, Michaela ^a  

^a UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

^b UNIVERSITY OF TEXAS AT DALLAS   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ATRIAL NATRIURETIC FACTOR; BRAIN NATRIURETIC PEPTIDE; GUANYLATE CYCLASE; NATRIURETIC PEPTIDE TYPE C;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLOOD PRESSURE MEASUREMENT; BLOOD PRESSURE REGULATION; CONTROLLED STUDY; ENZYME DEFICIENCY; HORMONAL REGULATION; HYPERTENSION; MOUSE; NONHUMAN; PEPTIDE ANALYSIS; PRIORITY JOURNAL; THORACIC AORTA; VASCULAR RING;

ANIMALS; AORTA; ATRIAL NATRIURETIC FACTOR; BLOOD PRESSURE; DOSE-RESPONSE RELATIONSHIP, DRUG; GUANYLATE CYCLASE; HYPERTENSION; MICE; MICE, MUTANT STRAINS; MUSCLE RELAXATION; NATRIURETIC PEPTIDE, BRAIN; NATRIURETIC PEPTIDE, C-TYPE; PROTEINS; RECEPTORS, ATRIAL NATRIURETIC FACTOR; SIGNAL TRANSDUCTION;

ANIMALIA;

EID: 0030760043     PISSN: 00219258     EISSN: None     Source Type: Journal    
DOI: 10.1074/jbc.272.37.23064     Document Type: Article

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