A genetic model defines the importance of the atrial natriuretic peptide receptor (guanylyl cyclase-A) in the regulation of kidney function

Proceedings of the National Academy of Sciences of the United States of America

Volumn 97, Issue 8, 2000, Pages 4369-4373

  Dubois, Susan K ^b,d   Kishimoto, Ichiro ^b,c,e   Lillis, Terence O ^d   Garbers, David L ^a,b,c  

^a UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

^b UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

^c UNIVERSITY OF TEXAS AT DALLAS   (United States)

^d WEST VIRGINIA UNIVERSITY SCHOOL OF MEDICINE   (United States)

^e KYOTO UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

ATRIAL NATRIURETIC FACTOR RECEPTOR; GUANYLATE CYCLASE;

ANIMAL EXPERIMENT; ARTICLE; CONTROLLED STUDY; DIURESIS; GENE CONTROL; GENE DISRUPTION; GENETIC MODEL; KIDNEY FUNCTION; MOUSE; NATRIURESIS; NONHUMAN; PRIORITY JOURNAL; REGULATORY MECHANISM; SODIUM EXCRETION;

ANIMALS; ATRIAL NATRIURETIC FACTOR; GUANYLATE CYCLASE; KIDNEY; MICE; MICE, KNOCKOUT; MODELS, GENETIC; RECEPTORS, ATRIAL NATRIURETIC FACTOR; SIGNAL TRANSDUCTION; SODIUM;

ANIMALIA; RODENTIA;

EID: 0034636044     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.97.8.4369     Document Type: Article

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