Signaling through CD14 attenuates the inflammatory response to Borrelia burgdorferi, the agent of lyme disease

Journal of Immunology

Volumn 174, Issue 3, 2005, Pages 1539-1548

  Benhnia, Mohammed Rafii El Idrissi ^a   Wroblewski, Danielle ^a   Akhtar, Muhammad Naveed ^a   Patel, Raina A ^a   Lavezzi, Wendy ^a   Gangloff, Sophie C ^b   Goyert, Sanna M ^c   Caimano, Melissa J ^d   Radolf, Justin D ^d   Sellati, Timothy J ^a  

^a ALBANY MEDICAL COLLEGE   (United States)

^b UNIVERSITÉ DE REIMS CHAMPAGNE ARDENNE   (France)

^c NEW YORK UNIVERSITY SCHOOL OF MEDICINE   (United States)

^d UNIVERSITY OF CONNECTICUT HEALTH CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BACTERIAL PROTEIN; CD14 ANTIGEN; CYTOKINE; LIPOPROTEIN; T LYMPHOCYTE RECEPTOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANTIGEN RECOGNITION; ARTICLE; BACTERIOLYSIS; BLOOD LEVEL; BORRELIA BURGDORFERI; CELL ACTIVATION; CHRONIC INFLAMMATION; CYTOKINE PRODUCTION; DISEASE SEVERITY; GENE CONTROL; IN VITRO STUDY; IN VIVO STUDY; INNATE IMMUNITY; LYME DISEASE; MACROPHAGE; MONOCYTE; MOUSE; NONHUMAN; POLYMORPHONUCLEAR CELL; PRIORITY JOURNAL; PROTEIN BINDING; SIGNAL TRANSDUCTION; T LYMPHOCYTE RECEPTOR GENE;

EID: 19944433179     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.174.3.1539     Document Type: Article

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