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Volumn 3, Issue 3, 2002, Pages 248-254
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Human MRE11 is inactivated in mismatch repair-deficient cancers
a a a a a a a a a a a a a |
Author keywords
[No Author keywords available]
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Indexed keywords
ATM PROTEIN;
MUTANT PROTEIN;
ONCOPROTEIN;
THYMINE;
ARTICLE;
ATAXIA TELANGIECTASIA;
CANCER CELL CULTURE;
CANCER SUSCEPTIBILITY;
CELL CYCLE S PHASE;
COLON CARCINOGENESIS;
COLORECTAL CANCER;
COLORECTAL TUMOR;
COMPLEX FORMATION;
CONTROLLED STUDY;
GENE EXPRESSION;
GENE FUNCTION;
GENE IDENTIFICATION;
GENE INACTIVATION;
GENE MUTATION;
GENE TARGETING;
HOMOZYGOSITY;
HUMAN;
HUMAN CELL;
INTRON;
IONIZING RADIATION;
MISMATCH REPAIR;
NIJMEGEN BREAKAGE SYNDROME;
NUCLEOTIDE SEQUENCE;
PRIORITY JOURNAL;
WILD TYPE;
AMINO ACID SEQUENCE;
BASE PAIR MISMATCH;
BASE SEQUENCE;
COLORECTAL NEOPLASMS;
DNA REPAIR;
DNA, NEOPLASM;
DNA-BINDING PROTEINS;
GENE SILENCING;
HUMANS;
MOLECULAR SEQUENCE DATA;
REPETITIVE SEQUENCES, NUCLEIC ACID;
RNA SPLICE SITES;
ATAXIA;
ATAXIA TELANGIECTASIA;
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EID: 18344395671
PISSN: 1469221X
EISSN: None
Source Type: Journal
DOI: 10.1093/embo-reports/kvf044 Document Type: Article |
Times cited : (162)
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References (26)
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