Mechanisms of disease: Advances in understanding the mechanisms leading to chronc pancreatitis

Nature Clinical Practice Gastroenterology and Hepatology

Volumn 1, Issue 1, 2004, Pages 46-52

  Whitcomb, David C ^a,b  

^a UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

^b PRESBYTERIAN UNIVERSITY HOSPITAL   (United States)

Author keywords

CFTR; Pancreatitis; SPINK1; Stellate cells; Trypsin

Indexed keywords

APROTININ; ARGININE; ASPARAGINE; CALCIUM; ISOLEUCINE; LIPID; LYSINE; ORGANOTIN COMPOUND; PARATHYROID HORMONE; PHENACETIN; TRANSMEMBRANE CONDUCTANCE REGULATOR; TRYPSIN; TRYPSINOGEN;

ACINAR CELL; ACUTE PANCREATITIS; ALCOHOL CONSUMPTION; AMINO ACID SUBSTITUTION; AUTOIMMUNITY; CALCIUM BINDING; CELL PROTECTION; CHRONIC KIDNEY FAILURE; CHRONIC PANCREATITIS; CIGARETTE SMOKING; DISEASE ASSOCIATION; DISEASE COURSE; DISEASE MODEL; DISEASE SEVERITY; ENVIRONMENTAL FACTOR; GENE MUTATION; GENETIC ANALYSIS; HEREDITY; HUMAN; HYPERCALCEMIA; HYPERLIPIDEMIA; HYPERPARATHYROIDISM; IMMUNOLOGY; MEDICAL RESEARCH; PATHOGENESIS; PATHOPHYSIOLOGY; PRIORITY JOURNAL; REVIEW; RISK ASSESSMENT; RISK FACTOR;

CHRONIC DISEASE; HUMANS; MODELS, BIOLOGICAL; PANCREATITIS; RISK FACTORS; TRYPSIN INHIBITORS; TRYPSINOGEN;

EID: 16244393935     PISSN: 17434378     EISSN: None     Source Type: Journal    
DOI: 10.1038/ncpgasthep0025     Document Type: Review

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