Gain-of-function mutations associated with hereditary pancreatitis enhance autoactivation of human cationic trypsinogen

Biochemical and Biophysical Research Communications

Volumn 278, Issue 2, 2000, Pages 286-289

  Sahin Tóth, Miklós ^a   Tóth, Miklós ^b  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b SEMMELWEIS UNIVERSITY   (Hungary)

Author keywords

[No Author keywords available]

Indexed keywords

ARGININE; ASPARAGINE; HISTIDINE; TRANSMEMBRANE CONDUCTANCE REGULATOR; TRYPSINOGEN;

ACINAR CELL; AMINO ACID SUBSTITUTION; ARTICLE; AUTOSOMAL DOMINANT DISORDER; CATALYSIS; ENZYME KINETICS; GENE FREQUENCY; GENE MUTATION; GENETIC DISORDER; GENETIC HETEROGENEITY; HUMAN; HUMAN CELL; PANCREATITIS; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN LOCALIZATION;

EID: 0034687560     PISSN: 0006291X     EISSN: None     Source Type: Journal    
DOI: 10.1006/bbrc.2000.3797     Document Type: Article

References (28)

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