Cellular Mechanisms of Contractile Dysfunction in Hibernating Myocardium

Circulation Research

Volumn 94, Issue 6, 2004, Pages 794-801

  Bito, Virginie ^a   Heinzel, Frank R ^a,d   Weidemann, Frank ^b   Dommke, Christophe ^b   Van Der Velden, Jolanda ^c   Verbeken, Erik ^b   Claus, Piet ^b   Bijnens, Bart ^b   De Scheerder, Ivan ^a   Stienen, Ger J M ^c   Sutherland, George R ^b   Sipido, Karin R ^a  

^a UNIVERSITY OF LEUVEN   (Belgium)

^b UNIVERSITY OF LEUVEN   (Belgium)

^c VU UNIVERSITY MEDICAL CENTER   (Netherlands)

^d UNIVERSITY OF DUISBURG ESSEN   (Germany)

Author keywords

Ca2+ current; Hibernation; Ion channels; Ischemia; Myocytes

Indexed keywords

CALCIUM; CALCIUM CHANNEL L TYPE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CALCIUM CURRENT; CALCIUM TRANSPORT; CONTROLLED STUDY; CORONARY ARTERY OBSTRUCTION; DISEASE SEVERITY; HEART CONTRACTION; HEART FAILURE; HEART MUSCLE CELL; HEART MUSCLE POTENTIAL; HEART MUSCLE REVASCULARIZATION; HEART VENTRICLE HYPERTROPHY; HIBERNATION; ISCHEMIC HEART DISEASE; NONHUMAN; PRIORITY JOURNAL; SWINE; VOLTAGE CLAMP;

ACTION POTENTIALS; ANIMALS; CALCIUM CHANNELS, L-TYPE; CALCIUM SIGNALING; CELL SIZE; CORONARY STENOSIS; HYPERTROPHY; ION TRANSPORT; MICROFILAMENTS; MYOCARDIAL CONTRACTION; MYOCARDIAL STUNNING; MYOCARDIUM; MYOCYTES, CARDIAC; NIFEDIPINE; PATCH-CLAMP TECHNIQUES; SWINE; TROPONIN I;

EID: 11144355700     PISSN: 00097330     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.RES.0000124934.84048.DF     Document Type: Article

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