Gap junctions regulate extracellular signal-regulated kinase signaling to affect gene transcription

Molecular Biology of the Cell

Volumn 16, Issue 1, 2005, Pages 64-72

  Stains, Joseph P ^a,b   Civitelli, Roberto ^a  

^a BARNES JEWISH HOSPITAL   (United States)

^b UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

COLLAGEN TYPE 1; COLLAGEN TYPE 1ALPHA1; CONNEXIN 43; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE KINASE; OSTEOCALCIN; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B; RAF PROTEIN; RAS PROTEIN; TRANSCRIPTION FACTOR SP1; UNCLASSIFIED DRUG;

ARTICLE; CELL COMMUNICATION; CONNEXIN RESPONSE ELEMENT; CONNEXIN45 GENE; CONTROLLED STUDY; DNA RESPONSIVE ELEMENT; GAP JUNCTION; GENE; GENE OVEREXPRESSION; GENETIC TRANSCRIPTION; HUMAN; HUMAN CELL; OSTEOBLAST; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN PHOSPHORYLATION; REGULATORY MECHANISM; SECOND MESSENGER; SIGNAL TRANSDUCTION; TRANSCRIPTION REGULATION;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ANIMALS; BLOTTING, WESTERN; CELL COMMUNICATION; CELL LINE, TUMOR; CELL MEMBRANE; CELL NUCLEUS; CHROMATIN IMMUNOPRECIPITATION; COLLAGEN TYPE I; CONNEXIN 43; CONNEXINS; EXTRACELLULAR SIGNAL-REGULATED MAP KINASES; FORMALDEHYDE; GAP JUNCTIONS; IMMUNOPRECIPITATION; LUCIFERASES; MODELS, BIOLOGICAL; OSTEOBLASTS; OSTEOCALCIN; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; PLASMIDS; PROTO-ONCOGENE PROTEINS C-RAF; RAS PROTEINS; RATS; RESPONSE ELEMENTS; SIGNAL TRANSDUCTION; SP1 TRANSCRIPTION FACTOR; TRANSCRIPTION, GENETIC; TRANSFECTION;

TRIXIS;

EID: 11144317306     PISSN: 10591524     EISSN: None     Source Type: Journal    
DOI: 10.1091/mbc.E04-04-0339     Document Type: Article

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