Pharmacological Modulation of Cardiac Gap Junctions to Enhance Cardiac Conduction: Evidence Supporting a Novel Target for Antiarrhythmic Therapy

Circulation

Volumn 108, Issue 25, 2003, Pages 3157-3163

  Eloff, Benjamin C ^a   Gilat, Eran ^a   Wan, Xiaoping ^a   Rosenbaum, David S ^a,b  

^a Case Western Reserve University   (United States)

^b CASE WESTERN RESERVE UNIVERSITY   (United States)

Author keywords

Electrophysiology; Ischemia; Mapping

Indexed keywords

ANTIARRHYTHMIC AGENT; PEPTIDE; ROTIGAPTIDE; UNCLASSIFIED DRUG;

ACIDOSIS; ANIMAL CELL; ANIMAL TISSUE; ARRHYTHMOGENESIS; ARTICLE; CELL ADHESION; CONTROLLED STUDY; DRUG TARGETING; EPICARDIUM; EVIDENCE BASED MEDICINE; GAP JUNCTION; GUINEA PIG; HEART CONDUCTION; HEART MUSCLE CELL; HEART MUSCLE ISCHEMIA; HEART MUSCLE POTENTIAL; HEART REPOLARIZATION; ISOLATED HEART; NONHUMAN; OPTICS; PRIORITY JOURNAL; SODIUM CURRENT;

ACTION POTENTIALS; ANIMALS; ANTI-ARRHYTHMIA AGENTS; ARRHYTHMIA; BODY SURFACE POTENTIAL MAPPING; CELLS, CULTURED; DRUG DELIVERY SYSTEMS; ELECTRIC CONDUCTIVITY; GAP JUNCTIONS; GUINEA PIGS; HEART; HYDROGEN-ION CONCENTRATION; MYOCYTES, CARDIAC; OLIGOPEPTIDES; ORGAN CULTURE TECHNIQUES;

EID: 0347418930     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.0000101926.43759.10     Document Type: Article

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