Contribution of sodium channel mutations to bradycardia and sinus node dysfunction in LQT3 families

Circulation Research

Volumn 92, Issue 9, 2003, Pages 976-983

  Veldkamp, Marieke W ^a   Wilders, Ronald ^a   Baartscheer, Antonius ^a   Zegers, Jan G ^a   Bezzina, Connie R ^a   Wilde, Arthur A M ^a  

^a UNIVERSITY OF AMSTERDAM   (Netherlands)

Author keywords

Electrophysiology; Ion channels; Long QT syndrome; Sinoatrial node; Sudden death

Indexed keywords

SODIUM CHANNEL;

ACTION POTENTIAL; ARTICLE; BRADYCARDIA; CLINICAL FEATURE; CONTROLLED STUDY; GENE MUTATION; HEART DEPOLARIZATION; HEART REPOLARIZATION; HUMAN; HUMAN CELL; LONG QT SYNDROME; PRIORITY JOURNAL; SINUS NODE; SINUS NODE DYSFUNCTION;

ACTION POTENTIALS; BRADYCARDIA; CELL LINE; COMPUTER SIMULATION; ELECTRIC CONDUCTIVITY; GENETIC PREDISPOSITION TO DISEASE; HUMANS; LONG QT SYNDROME; MUTATION; PATCH-CLAMP TECHNIQUES; SINOATRIAL NODE; SODIUM CHANNELS;

EID: 0037539913     PISSN: 00097330     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.RES.0000069689.09869.A8     Document Type: Article

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