Na+ channel mutation that causes both Brugada and long-QT syndrome phenotypes: A simulation study of mechanism

Circulation

Volumn 105, Issue 10, 2002, Pages 1208-1213

  Clancy, Colleen E ^a   Rudy, Yoram ^a,b  

^a Case Western Reserve University   (United States)

^b CASE WESTERN RESERVE UNIVERSITY   (United States)

Author keywords

Arrhythmia; Brugada syndrome; Genes; Long QT syndrome; Sodium

Indexed keywords

SODIUM CHANNEL;

ARRHYTHMOGENESIS; ARTICLE; BRUGADA SYNDROME; COMPUTER MODEL; COMPUTER SIMULATION; CONTROLLED STUDY; GENE MUTATION; HUMAN; HUMAN CELL; LONG QT SYNDROME; PRIORITY JOURNAL;

ACTION POTENTIALS; ARRHYTHMIA; COMPUTER SIMULATION; ELECTROCARDIOGRAPHY; HEART VENTRICLES; HUMANS; INTERNET; ION CHANNEL GATING; LONG QT SYNDROME; MARKOV CHAINS; MODELS, CARDIOVASCULAR; MUTATION; MYOCARDIUM; PHENOTYPE; REPRODUCIBILITY OF RESULTS; SODIUM; SODIUM CHANNELS; STRUCTURE-ACTIVITY RELATIONSHIP;

EID: 0037066036     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/hc1002.105183     Document Type: Article

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