Arrhythmogenic mechanism of an LQT-3 mutation of the human heart Na+ channel α-subunit: A comptutational analysis

Circulation

Volumn 102, Issue 5, 2000, Pages 584-590

  Wehrens, X H T ^a   Abriel, H ^a   Cabo, C ^a   Benhorin, J ^b   Kass, R S ^a  

^a Columbia University ^*  (United States)

^b BIKUR CHOLIM HOSPITAL   (Israel)

Author keywords

Action potentials; Arrhythmia; Calcium; Sodium; Torsad de pointes

Indexed keywords

SODIUM CHANNEL;

ARRHYTHMOGENESIS; ARTICLE; CALCIUM CELL LEVEL; HEART RATE; HUMAN; HUMAN CELL; LONG QT SYNDROME; MEMBRANE CURRENT; MUSCLE ACTION POTENTIAL; PHENOTYPE; PRIORITY JOURNAL;

EID: 0034255466     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.102.5.584     Document Type: Article

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