Transcriptional activators differ in their abilities to control alternative splicing

Journal of Biological Chemistry

Volumn 277, Issue 45, 2002, Pages 43110-43114

  Nogués, Guadalupe ^a   Kadener, Sebastián ^a,c   Cramer, Paula ^a   Bentley, David ^a   Kornblihtt, Alberto R ^b  

^a INSTITUTO DE FISIOLOGÍA   (Argentina)

^b UNIVERSITY OF COLORADO   (United States)

^c HARVARD UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ENZYME INHIBITION; ENZYMES; GENES; RNA; VIRUSES;

TRANSCRIPTIONAL ACTIVATORS;

BIOCHEMISTRY;

5,6 DICHLOROBENZIMIDAZOLE RIBOSIDE; FIBRONECTIN; HISTONE DEACETYLASE INHIBITOR; MUTANT PROTEIN; TRANSACTIVATOR PROTEIN; TRANSCRIPTION FACTOR SP1; TRICHOSTATIN A;

ALTERNATIVE RNA SPLICING; ANIMAL CELL; ARTICLE; CORRELATION ANALYSIS; ENHANCER REGION; EXON; NONHUMAN; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN DOMAIN; PROTEIN FOLDING;

ALTERNATIVE SPLICING; ANIMALS; ANTIGENS, POLYOMAVIRUS TRANSFORMING; CERCOPITHECUS AETHIOPS; COS CELLS; ENHANCER ELEMENTS (GENETICS); EXONS; FIBRONECTINS; GENE EXPRESSION REGULATION, VIRAL; REPLICATION ORIGIN; RNA POLYMERASE II; SIMIAN VIRUS 40; TRANS-ACTIVATORS; TRANSCRIPTION, GENETIC; TRANSFECTION;

ANIMALIA; HUMAN IMMUNODEFICIENCY VIRUS;

EID: 0037044769     PISSN: 00219258     EISSN: None     Source Type: Journal    
DOI: 10.1074/jbc.M208418200     Document Type: Article

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