Effect of the first window of ischemic preconditioning on mitochondrial dysfunction following global cerebral ischemia

Mitochondrion

Volumn 2, Issue 3, 2002, Pages 181-189

  Pérez Pinzón, Miguel A ^a   Basit, Abdul ^a   Dave, Kunjan R ^a   Busto, Raul ^a   Veauvy, Clemence ^a   Saul, Isabel ^a   Ginsberg, Myron D ^a   Sick, Thomas J ^a  

^a UNIVERSITY OF MIAMI MILLER SCHOOL OF MEDICINE   (United States)

Author keywords

Anoxia; Cell death; Free radicals; Tolerance

Indexed keywords

ASCORBIC ACID; MALIC ACID; PHENYLENEDIAMINE DERIVATIVE; PYRUVIC ACID; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); ROTENONE; SUCCINATE DEHYDROGENASE (UBIQUINONE); SUCCINIC ACID DERIVATIVE; UBIQUINOL CYTOCHROME C REDUCTASE; UBIQUINONE DERIVATIVE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANOXIA; ARTICLE; BRAIN ISCHEMIA; BRAIN MITOCHONDRION; BRAIN SLICE; BREATHING RATE; CAROTID ARTERY OBSTRUCTION; CELL ACTIVITY; CELL FUNCTION; CONTROLLED STUDY; DISEASE ACTIVITY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; HIPPOCAMPUS; HYPOTENSION; HYPOTHESIS; MITOCHONDRIAL RESPIRATION; NEUROPROTECTION; NONHUMAN; OXIDATIVE PHOSPHORYLATION; PRIORITY JOURNAL; PROTEIN FUNCTION; RAT; REPERFUSION;

EID: 0036889404     PISSN: 15677249     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1567-7249(02)00070-3     Document Type: Article

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