Pyruvate improves redox status and decreases indicators of hepatic apoptosis during hemorrhagic shock in swine

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 283, Issue 4 52-4, 2002, Pages

  Mongan, Paul D ^a,b   Capacchione, John ^a   West, Shanda ^a   Karaian, John ^a   Dubois, Dawn ^a   Keneally, Ryan ^a   Sharma, Pushpa ^a  

^a Uniformed Services University of the Health Sciences and the Walter Reed National Military Medical Center   (United States)

^b UNIFORMED SERVICES UNIVERSITY OF THE HEALTH SCIENCES   (United States)

Author keywords

Caspases; Glutathione; Redox state

Indexed keywords

CASPASE; CYTOCHROME C; GLUTATHIONE; HALOTHANE; LACTIC ACID; NICOTINAMIDE ADENINE DINUCLEOTIDE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; PROTEIN BAX; PROTEIN BCL X; PYRUVIC ACID; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE;

ACID BASE BALANCE; ADOLESCENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; ENZYME DEGRADATION; HEMODYNAMICS; HEMORRHAGIC SHOCK; LIVER INJURY; LIVER MITOCHONDRION; LIVER PROTECTION; MICRODIALYSIS; NONHUMAN; OSMOLALITY; OXIDATION REDUCTION STATE; PRIORITY JOURNAL; SWINE;

EID: 0036783927     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.01073.2001     Document Type: Article

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