Chemokines, inflammation and Trypanosoma cruzi infection

Trends in Parasitology

Volumn 18, Issue 6, 2002, Pages 262-265

  Teixeira, Mauro M ^a   Gazzinelli, Ricardo T ^a,b   Silva, João S ^c  

^a FEDERAL UNIVERSITY OF MINAS GERAIS   (Brazil)

^b INSTITUTO OSWALDO CRUZ   (Brazil)

^c UNIVERSITY OF SÃO PAULO   (Brazil)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA CHEMOKINE; CHEMOKINE; CHEMOKINE RECEPTOR CCR5; GAMMA INTERFERON; INTERLEUKIN 10; INTERLEUKIN 1BETA; MACROPHAGE INFLAMMATORY PROTEIN 1ALPHA; MACROPHAGE INFLAMMATORY PROTEIN 1BETA; MONOCYTE CHEMOTACTIC PROTEIN 1; RANTES; TRANSFORMING GROWTH FACTOR BETA; TUMOR NECROSIS FACTOR ALPHA;

CHAGAS DISEASE; CYTOKINE PRODUCTION; GROWTH; HOST RESISTANCE; HUMAN; IMMUNE RESPONSE; IMMUNOCOMPETENT CELL; INFECTION RESISTANCE; INFLAMMATION; INFLAMMATORY CELL; NEUTROPHIL CHEMOTAXIS; NONHUMAN; PATHOGENESIS; REVIEW; TRYPANOSOMA CRUZI; TRYPANOSOMIASIS;

ANIMALS; CHAGAS DISEASE; CHEMOKINES; HUMANS; MICE; MICE, INBRED C3H; MICE, INBRED C57BL; TRYPANOSOMA CRUZI;

PROTOZOA; TRYPANOSOMA; TRYPANOSOMA CRUZI;

EID: 0036605949     PISSN: 14714922     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1471-4922(02)02283-3     Document Type: Review

References (25)

1. Parasite persistence in the aetiology of Chagas disease
2. Autoimmunity in Chagas heart disease
3. Immunological control of Trypanosoma cruzi infection and pathogenesis of Chagas disease
4. Chemokines: Immunology's high impact factors
5. Cecilio Romana, Romana's sign and Chagas' disease
6. Proinflammatory activity of glycosylphosphatidylinositol anchors derived from Trypanosoma cruzi: Structural and functional analyses
7. DNA from protozoan parasites Babesia bovis, Trypanosoma cruzi, and T. brucei is mitogenic for B lymphocytes and stimulates macrophage expression of interleukin-12, tumor necrosis factor-α, and nitric oxide
8. Beta-Chemokines enhance parasite uptake and promote nitric oxide-dependent microbiostatic activity in murine inflammatory macrophages infected with Trypanosoma cruzi
9. Kinetics of cytokine gene expression in experimental chagasic cardiomyopathy: Tissue parasitism and endogenous IFN-γ as important determinants of chemokine mRNA expression during infection with Trypanosoma cruzi
10. Glycosylphosphatidylinositol-anchored mucin-like glycoproteins isolated from Trypanosoma cruzi trypomastigotes induce in vivo leukocyte recruitment dependent on MCP-1 production by IFN-γ-primed macrophages
11. Trypanosoma cruzi-infected cardiomyocytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity
12. Modulation of chemokine production and inflammatory responses in interferon-γ- and tumor necrosis factor-R1-deficient mice during Trypanosoma cruzi infection
13. Lymphocyte traffic control by chemokines
14. High activation phenotype and expression of IFN-γ-inducible adhesion and chemoattractant molecules
15. + lymphocytes
16. + T cells with the presence of Trypanosoma cruzi antigens in chronic, human, chagasic myocarditis
17. Susceptibility of beta 2-microglobulin-deficient mice to Trypanosoma cruzi infection
18. Activation of Toll-like receptor-2 by glycosylphosphatidylinositol anchors from a protozoan parasite
19. The cysteine-cysteine family of chemokines RANTES, MIP-1α, and MIP-1β induce trypanocidal activity in human macrophages via nitric oxide
20. Beta-chemokines that inhibit HIV-1 infection of human macrophages stimulate uptake and promote destruction of Trypanosoma cruzi by human macrophages
21. Chemokine receptor CCR5 polymorphisms and Chagas' disease cardiomyopathy
22. PCR detection of Trypanosoma cruzi DNA in oesophageal tissues of patients with chronic digestive Chagas' disease
23. The pathogenesis of Chagas' disease: When autoimmune and parasite-specific immune responses meet
24. Platelet-activating factor induces nitric oxide synthesis in Trypanosoma cruzi-infected macrophages and mediates resistance to parasite infection in mice
25. 4 induces nitric oxide synthesis in Trypanosoma cruzi-infected murine macrophages and mediates resistance to infection in mice