Exposure to a dysfunctional glucocorticoid receptor from early embryonic life programs the resistance to experimental autoimmune encephalomyelitis via nitric oxide-induced immunosuppression

Journal of Immunology

Volumn 168, Issue 11, 2002, Pages 5848-5859

  Marchetti, Bianca ^a   Morale, Maria C ^a   Brouwer, Jantien ^a   Tirolo, Cataldo ^a   Testa, Nuccio ^a   Caniglia, Salvo ^a   Barden, Nicholas ^a   Amor, Sandra ^a   Smith, Paul A ^a   Dijkstra, Christine D ^a  

^a OASI RESEARCH INSTITUTE IRCCS   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

COMPLEMENTARY RNA; GLUCOCORTICOID; GLUCOCORTICOID RECEPTOR; GLYCOPROTEIN; MYELIN; N6 (1 IMINOETHYL)LYSINE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE INHIBITOR; NITRITE;

ALLERGIC ENCEPHALOMYELITIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL PROLIFERATION; CONTROLLED STUDY; DISEASE PREDISPOSITION; DOWN REGULATION; EFFECTOR CELL; FEMALE; GENE EXPRESSION; GENE INDUCTION; HYPOTHALAMUS HYPOPHYSIS ADRENAL SYSTEM; IMMUNE DEFICIENCY; LYMPH NODE CELL; MALE; MOUSE; MULTIPLE SCLEROSIS; NONHUMAN; OLIGODENDROGLIA; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; SPLEEN CELL; TRANSGENIC MOUSE;

EID: 0036604085     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.168.11.5848     Document Type: Article

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