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Volumn 362, Issue 2, 2002, Pages 443-451
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Two mutations in troponin I that cause hypertrophic cardiomyopathy have contrasting effects on cardiac muscle contractility
a a a a a a a a |
Author keywords
Calcium; Motility; Regulation
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Indexed keywords
ADENOSINETRIPHOSPHATE;
BIOASSAY;
ESCHERICHIA COLI;
FIBERS;
MUSCLE;
PROTEINS;
MUTATIONS;
CARDIOLOGY;
ACTIN;
ARGININE;
CALCIUM ION;
GLYCINE;
MYOSIN ADENOSINE TRIPHOSPHATASE;
SERINE;
TROPOMYOSIN;
TROPONIN C;
TROPONIN I;
VANADIC ACID;
ANIMAL TISSUE;
ARTICLE;
CONTROLLED STUDY;
ESCHERICHIA COLI;
GUINEA PIG;
HEART MUSCLE CONTRACTILITY;
HYPERTROPHIC CARDIOMYOPATHY;
MUTATION;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN CONFORMATION;
WESTERN BLOTTING;
ACTINS;
AMINO ACID SUBSTITUTION;
ANIMALS;
CALCIUM;
CARDIOMYOPATHY, HYPERTROPHIC;
GUINEA PIGS;
HEART;
HUMANS;
MUSCLE FIBERS;
MUTATION, MISSENSE;
MYOCARDIAL CONTRACTION;
MYOSIN SUBFRAGMENTS;
PHENOTYPE;
TROPOMYOSIN;
TROPONIN I;
ANIMALIA;
CAVIA PORCELLUS;
ESCHERICHIA COLI;
ORYCTOLAGUS CUNICULUS;
SUS SCROFA;
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EID: 0036499710
PISSN: 02646021
EISSN: None
Source Type: Journal
DOI: 10.1042/0264-6021:3620443 Document Type: Article |
Times cited : (34)
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References (39)
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