JunB can substitute for Jun in mouse development and cell proliferation

Nature Genetics

Volumn 30, Issue 2, 2002, Pages 158-166

  Passegué, Emmanuelle ^a,b   Jochum, Wolfram ^a,c   Behrens, Axel ^a,d   Ricci, Romeo ^a   Wagne, Erwin F ^a  

^a RESEARCH INSTITUTE OF MOLECULAR PATHOLOGY   (Austria)

^b STANFORD UNIVERSITY   (United States)

^c UNIVERSITY HOSPITAL ZURICH   (Switzerland)

^d IMPERIAL CANCER RESEARCH FUND   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

GENE PRODUCT; PROTEIN FOS; TRANSCRIPTION FACTOR AP 1; TRANSCRIPTION FACTOR JUNB; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL MATURATION; CELL PROLIFERATION; CONTROLLED STUDY; FIBROBLAST; GENE EXPRESSION REGULATION; IN VITRO STUDY; LIVER DEVELOPMENT; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; SEQUENCE ANALYSIS; TRANSCRIPTION REGULATION; TRANSGENIC MOUSE;

ANIMALS; CELL DIVISION; CYCLIN D1; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; EMBRYONIC AND FETAL DEVELOPMENT; GENE EXPRESSION REGULATION, DEVELOPMENTAL; GENES, FOS; GENES, JUN; HEART DEFECTS, CONGENITAL; LIVER; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MICE, TRANSGENIC; PHENOTYPE; PROTO-ONCOGENE PROTEINS C-JUN; TRANSCRIPTION FACTOR AP-1; TUMOR SUPPRESSOR PROTEIN P53;

ANIMALIA; MUS MUSCULUS;

EID: 0036463679     PISSN: 10614036     EISSN: None     Source Type: Journal    
DOI: 10.1038/ng790     Document Type: Article

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