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Volumn 129, Issue 1-2, 2002, Pages 97-105

Truncation of the neuritogenic peptide bP2(60-70) results in the generation of altered peptide ligands with the potential to interfere with T cell activation

Author keywords

APL; EAE, experimental autoimmune encephalomyelitis; EAN; Signaling; T cells

Indexed keywords

CD3 ANTIGEN; GAMMA INTERFERON; LIGAND; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 2; MITOGEN ACTIVATED PROTEIN KINASE; MYELIN BASIC PROTEIN; PEPTIDE; PEPTIDE BP2[60-69]; PEPTIDE BP2[60-70]; PEPTIDE BP2[61-69]; PEPTIDE BP2[61-70]; PEPTIDE BP2[62-68]; PEPTIDE BP2[62-69]; PEPTIDE BP2[63-69]; T LYMPHOCYTE RECEPTOR; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG;

EID: 0036346759     PISSN: 01655728     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0165-5728(02)00183-2     Document Type: Article
Times cited : (5)

References (42)
  • 42


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.