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Volumn 14, Issue 4, 2001, Pages 585-594

Exogenous nitric oxide causes potentiation of hippocampal synaptic transmission during low-frequency stimulation via the endogenous nitric oxide-cGMP pathway

Author keywords

Hippocampus; Long term potentiation; Nitric oxide; Rat; Soluble guanylyl cyclase; Synaptic plasticity

Indexed keywords

1,1 DIETHYL 2 HYDROXY 2 NITROSOHYDRAZINE SODIUM; 1H 1,2,4 OXADIAZOLO[4,3 A]QUINOXALIN 1 ONE; CYCLIC GMP; GUANYLATE CYCLASE; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; N METHYL DEXTRO ASPARTIC ACID RECEPTOR BLOCKING AGENT; N(G) NITROARGININE; NITRIC OXIDE; NITRIC OXIDE DONOR; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE INHIBITOR; UNCLASSIFIED DRUG; 1,1-DIETHYL-2-HYDROXY-2-NITROSOHYDRAZINE; 1H-(1,2,4)OXADIAZOLO(4,3-A)QUINOXALIN-1-ONE; 2 AMINO 5 PHOSPHONOVALERIC ACID; ADENOSINE TRIPHOSPHATE; CYANIDE; ENZYME INHIBITOR; HYDRAZINE DERIVATIVE; NITROGEN OXIDE; OXADIAZOLE DERIVATIVE; QUINOXALINE DERIVATIVE;

EID: 0036283453     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.0953-816X.2001.01680.x     Document Type: Article
Times cited : (44)

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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.