Short communication nitric oxide signalling is required for the generation of anoxia-induced long-term potentiation in the hippocampus

European Journal of Neuroscience

Volumn 9, Issue 10, 1997, Pages 2202-2206

  Huang, Chiung Chun ^a  

^a NATIONAL CHENG KUNG UNIVERSITY   (Taiwan)

Author keywords

Anoxie ltp; Hippocampus; Ischaemia; Nitric oxide

Indexed keywords

7 NITROINDAZOLE; ARGININE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE INHIBITOR;

ANIMAL TISSUE; ARTICLE; BRAIN HYPOXIA; BRAIN SLICE; CONTROLLED STUDY; DRUG EFFECT; EXCITATORY POSTSYNAPTIC POTENTIAL; HIPPOCAMPUS; INTRACELLULAR RECORDING; LONG TERM POTENTIATION; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; SYNAPTIC TRANSMISSION;

ANIMALS; ANOXIA; ARGININE; ENZYME INHIBITORS; EXCITATORY POSTSYNAPTIC POTENTIALS; HIPPOCAMPUS; INDAZOLES; LONG-TERM POTENTIATION; MALE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; NITROARGININE; RATS; RATS, SPRAGUE-DAWLEY; REACTION TIME; SIGNAL TRANSDUCTION; SYNAPTIC TRANSMISSION;

EID: 0030694844     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1460-9568.1997.tb01387.x     Document Type: Article

References (3)

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2. Bohme, G. A., Bon, C., Stutzmann, J. M., Doble, A. and Balanchard, J. C. (1991) Possible involvement of nitric oxide in long-term potentiation. Eur. J. Pharmacol., 199, 379-381.
3. Buisson, A., Margaill, I., Callebert, J., Plotkine, M. and Boulu, R. G. (1993) Mechanisms involved in the neuroprotective activity of a nitric oxide synthase inhibitor during focal cerebral ischemia. J. Neurochem., 61, 690-698.