Functional analysis of the Helicobacter pylori cag pathogenicity island reveals both VirD4-CagA-dependent and VirD4-CagA-independent mechanisms

Infection and Immunity

Volumn 70, Issue 2, 2002, Pages 665-671

  Selbach, Matthias ^a   Moese, Stefan ^a   Meyer, Thomas F ^a   Backert, Steffen ^a  

^a MAX PLANCK INSTITUTE FOR INFECTION BIOLOGY   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

CAGA PROTEIN; INTERLEUKIN 8;

ARTICLE; CYTOKINE RELEASE; GENE EXPRESSION; GENE FUNCTION; GENE TRANSLOCATION; HELICOBACTER INFECTION; HELICOBACTER PYLORI; IMMUNE RESPONSE; NONHUMAN; PATHOGENICITY; PHENOTYPE; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; PROTEIN SECRETION; STOMACH EPITHELIUM;

ANTIGENS, BACTERIAL; BACTERIAL PROTEINS; CULTURE MEDIA; GENES, BACTERIAL; HELICOBACTER PYLORI; HUMANS; INTERLEUKIN-8; PHOSPHORYLATION; TUMOR CELLS, CULTURED; VIRULENCE FACTORS;

EID: 0036156209     PISSN: 00199567     EISSN: None     Source Type: Journal    
DOI: 10.1128/IAI.70.2.665-671.2002     Document Type: Article

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