The extracellular regulated kinases (ERK) 1/2 mediate cannabinoid-induced inhibition of gap junctional communication in endothelial cells

British Journal of Pharmacology

Volumn 136, Issue 5, 2002, Pages 709-716

  Brandes, R P ^a   Popp, R ^a   Ott, G ^a   Bredenkotter D ^a   Wallner, C ^a   Busse, R ^a   Fleming, I ^a  

^a UNIVERSITY HOSPITAL FRANKFURT   (Germany)

Author keywords

Cannabinoid; Connexin; EDHF; Endothelium derived hyperpolarizing factor; Gap junctions; Map kinases

Indexed keywords

1,4 DIAMINO 1,4 BIS(2 AMINOPHENYLTHIO) 2,3 DICYANOBUTADIENE; 2 (2 AMINO 3 METHOXYPHENYL)CHROMONE; 2 [1 (3 AMIDINOTHIOPROPYL) 1H INDOL 3 YL] 3 (1 METHYL 1H INDOL 3 YL)MALEIMIDE; 4 (4 FLUOROPHENYL) 2 (4 METHYLSULFINYLPHENYL) 5 (4 PYRIDYL)IMIDAZOLE; BRADYKININ; CONNEXIN 43; DEXANABINOL; DICLOFENAC; DRONABINOL; MITOGEN ACTIVATED PROTEIN KINASE 1; N(G) NITROARGININE; NITRIC OXIDE; PROSTACYCLIN; RIMONABANT; BIOLOGICAL FACTOR; CANNABINOID; DRUG DERIVATIVE; ENDOTHELIUM DEPENDENT HYPERPOLARIZATION FACTOR; ENDOTHELIUM-DEPENDENT HYPERPOLARIZATION FACTOR; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE 3; TETRAHYDROCANNABINOL;

ARTERY DILATATION; ARTICLE; CELL COMMUNICATION; CONTROLLED STUDY; CORONARY ARTERY; DRUG EFFECT; ENDOTHELIUM CELL; ENZYME ACTIVATION; ENZYME PHOSPHORYLATION; GAP JUNCTION; HUMAN; HUMAN CELL; HYPERPOLARIZATION; MEMBRANE POTENTIAL; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; ANIMAL; CELL JUNCTION; ENZYMOLOGY; IN VITRO STUDY; METABOLISM; NERVE CELL INHIBITION; PHYSIOLOGY; SWINE; VASCULAR ENDOTHELIUM;

ANIMALS; BIOLOGICAL FACTORS; CANNABINOIDS; ENDOTHELIUM, VASCULAR; GAP JUNCTIONS; HUMANS; MITOGEN-ACTIVATED PROTEIN KINASE 1; MITOGEN-ACTIVATED PROTEIN KINASE 3; MITOGEN-ACTIVATED PROTEIN KINASES; NEURAL INHIBITION; SWINE; TETRAHYDROCANNABINOL;

EID: 0035987781     PISSN: 00071188     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.bjp.0704776     Document Type: Article

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