Do all voltage-gated potassium channels use MiRPs?

Circulation Research

Volumn 88, Issue 10, 2001, Pages 981-983

  Abbott, Geoffrey W ^a   Goldstein, Steve A N ^a,b   Sesti, Federico ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b NONE   (United States)

Author keywords

IKr; Ito; KCNE2; MiRP1; Potassium channels

Indexed keywords

MINK RELATED PEPTIDE; POTASSIUM CHANNEL; PROTEIN; UNCLASSIFIED DRUG;

CHANNEL GATING; COMPLEX FORMATION; EDITORIAL; HUMAN; NONHUMAN; PRIORITY JOURNAL; PROTEIN ASSEMBLY; PROTEIN EXPRESSION; PROTEIN LOCALIZATION;

EID: 0035947743     PISSN: 00097330     EISSN: None     Source Type: Journal    
DOI: 10.1161/hh1001.091869     Document Type: Editorial

References (25)

1. MinK-related peptide 1 associates with Kv4.2 and modulates its gating function: Potential role as β subunit of cardiac transient outward channel?
2. MiRP1 forms IKr potassium channels with HERG and is associated with cardiac arrhythmia
3. A common polymorphism associated with antibiotic-induced cardiac arrhythmia
4. MinK subdomains that mediate modulation of and association with KvLQT1
5. + channel by dual pathways
6. Analysis of the cyclic nucleotide-binding domain of the HERG potassium channel and interactions with KCNE2
7. A constitutively open potassium channel formed by KCNQ1 and KCNE3
8. MiRP2 forms potassium channels in skeletal muscle with Kv3.4 and is associated with periodic paralysis
9. A superfamily of small potassium channel subunits: Form and function of the MinK-related peptides (MiRPs)
10. The KCNE superfamily of potassium channel subunits: Physiology and pathophysiology of the MinK-related peptides (MiRPs)
11. Coassembly of K(V)Lqt1 and MinK (Isk) proteins to form cardiac I-Ks potassium channel
12. K(V)LQT1 and IsK (MinK) proteins associate to form the I(Ks) cardiac potassium current
13. Cloning of a membrane protein that induces a slow voltage-gated potassium current
14. Are Xenopus oocytes unique in displaying functional IsK channel heterologous expression?
15. Single-channel characteristics of wildtype IKs channels and channels formed with two MinK mutants that cause long QT syndrome
16. - channels
17. Induction of endogenous channels by high levels of heterologous membrane proteins in Xenopus oocytes
18. Viral and cellular small integral membrane proteins can modify ion channels endogenous to Xenopus oocytes
19. Long QT syndrome-associated mutations in the S4-S5 linker of KvLQT1 potassium channels modify gating and interaction with MinK subunits
20. Distinct gene-specific mechanisms of arrhythmia revealed by cardiac gene transfer of two long QT disease genes, HERG and KCNE1
21. A role for MiRP1-Kv4.2 in CNS A-type currents?
22. Ko modulation of HERG deactivation involves multiple channel domains and is affected by hMiRP1
23. + conduction and selectivity
24. Subunit composition of MinK potassium channels
25. MinK-KvLQT1 fusion proteins, evidence for multiple stoichiometries of the assembled IsK channel