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Volumn 61, Issue 12, 2001, Pages 4756-4760
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Activated granulocytes and granulocyte-derived hydrogen peroxide are the underlying mechanism of suppression of T-cell function in advanced cancer patients
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Author keywords
[No Author keywords available]
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Indexed keywords
CYTOKINE;
HYDROGEN PEROXIDE;
T LYMPHOCYTE RECEPTOR;
ARTICLE;
CANCER PATIENT;
CONTROLLED STUDY;
CYTOKINE PRODUCTION;
DENSITY GRADIENT;
GRANULOCYTE;
HUMAN;
HUMAN CELL;
IMMUNOSUPPRESSIVE TREATMENT;
IMMUNOTHERAPY;
LIPID PEROXIDATION;
MONONUCLEAR CELL;
OXIDATIVE STRESS;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
RESPIRATORY BURST;
T LYMPHOCYTE;
T LYMPHOCYTE ACTIVATION;
ADENOCARCINOMA;
BREAST NEOPLASMS;
COLONIC NEOPLASMS;
GRANULOCYTES;
HUMANS;
HYDROGEN PEROXIDE;
INTERFERON TYPE II;
INTERLEUKIN-2;
INTERLEUKIN-4;
MEMBRANE PROTEINS;
N-FORMYLMETHIONINE LEUCYL-PHENYLALANINE;
NEOPLASMS;
OXIDATIVE STRESS;
PANCREATIC NEOPLASMS;
RECEPTORS, ANTIGEN, T-CELL;
T-LYMPHOCYTES;
TUMOR NECROSIS FACTOR-ALPHA;
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EID: 0035874990
PISSN: 00085472
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
Times cited : (789)
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References (33)
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