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Volumn 20, Issue 7, 2001, Pages 859-868
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Inhibition of NF-κB sensitizes human pancreatic carcinoma cells to apoptosis induced by etoposide (VP16) or doxorubicin
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Author keywords
Anthracyclin; Chemotherapy; DNA damage; Phyllotoxin; Programmed cell death; Transcription factor
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Indexed keywords
ANTHRACYCLINE;
ANTINEOPLASTIC AGENT;
BENZYLOXYCARBONYLLEUCYLLEUCYLLEUCINAL;
DOXORUBICIN;
ETOPOSIDE;
GLIOTOXIN;
GYRASE INHIBITOR;
I KAPPA B;
IMMUNOGLOBULIN ENHANCER BINDING PROTEIN;
PROTEIN INHIBITOR;
SALAZOSULFAPYRIDINE;
BENZYLOXYCARBONYLLEUCYL LEUCYL LEUCINE ALDEHYDE;
BENZYLOXYCARBONYLLEUCYL-LEUCYL-LEUCINE ALDEHYDE;
DNA BINDING PROTEIN;
LEUPEPTIN;
NF KAPPAB INHIBITOR ALPHA;
NF-KAPPAB INHIBITOR ALPHA;
APOPTOSIS;
ARTICLE;
CANCER CHEMOTHERAPY;
CARCINOMA CELL;
CELL STRAIN;
DNA DAMAGE;
DRUG EFFECT;
DRUG EFFICACY;
DRUG RESPONSE;
DRUG SENSITIVITY;
GENETIC TRANSFECTION;
HUMAN;
HUMAN CELL;
PANCREAS CARCINOMA;
PRIORITY JOURNAL;
BIOSYNTHESIS;
CARCINOMA;
DRUG ANTAGONISM;
DRUG RESISTANCE;
PANCREAS TUMOR;
ANTINEOPLASTIC AGENTS;
CARCINOMA;
DNA-BINDING PROTEINS;
DOXORUBICIN;
DRUG RESISTANCE;
ETOPOSIDE;
GLIOTOXIN;
HUMANS;
I-KAPPA B PROTEINS;
LEUPEPTINS;
NF-KAPPA B;
PANCREATIC NEOPLASMS;
SULFASALAZINE;
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EID: 0035864344
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1204168 Document Type: Article |
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Times cited : (228)
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References (31)
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