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Oncogene

Volumn 20, Issue 46, 2001, Pages 6731-6741

Loss of p16 pathways stabilizes EWS/FLI1 expression and complements EWS/FLI1 mediated transformation

(2) Deneen, Benjamin a Denny, Christopher T a,b

a UNIVERSITY OF CALIFORNIA (United States)

b UNIVERSITY OF CALIFORNIA (United States)

Author keywords

Apoptosis; EWS FLI1; p16INK4a; p19ARF; SV40 large T antigen

Indexed keywords

PROTEIN P16; VIRUS LARGE T ANTIGEN;

APOPTOSIS; ARTICLE; EWING SARCOMA; HUMAN; HUMAN CELL; MALIGNANT TRANSFORMATION; NEUROECTODERM TUMOR; NONHUMAN; PRIORITY JOURNAL; RETROVIRUS; SIMIAN VIRUS 40; TUMOR SUPPRESSOR GENE;

3T3 CELLS; ANIMALS; APOPTOSIS; BLOTTING, NORTHERN; CELL DIVISION; CELL TRANSFORMATION, NEOPLASTIC; CYCLIN-DEPENDENT KINASE INHIBITOR P16; FIBROBLASTS; GENE DELETION; GREEN FLUORESCENT PROTEINS; HOMOZYGOTE; IMMUNOBLOTTING; KINETICS; LUMINESCENT PROTEINS; MICE; MICE, SCID; MODELS, BIOLOGICAL; MUTATION; ONCOGENE PROTEINS, FUSION; RETROVIRIDAE; TIME FACTORS; TRANSCRIPTION FACTORS; TRANSDUCTION, GENETIC; TUMOR SUPPRESSOR PROTEIN P14ARF; TUMOR SUPPRESSOR PROTEIN P53;

EID: 0035845859 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204875 Document Type: Article

Times cited : (120)

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