|
Volumn 167, Issue 5, 2001, Pages 2985-2990
|
Deficiency in β2-microglobulin, but not CD1, accelerates spontaneous lupus skin disease while inhibiting nephritis in MRL-Faslpr mice: An example of disease regulation at the organ level
a a a a a a |
Author keywords
[No Author keywords available]
|
Indexed keywords
BETA 2 MICROGLOBULIN;
FAS ANTIBODY;
IMMUNOGLOBULIN G;
MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 1;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
AUTOIMMUNITY;
CELL COMPARTMENTALIZATION;
CONTROLLED STUDY;
GENE TARGETING;
LUPUS ERYTHEMATOSUS NEPHRITIS;
LYMPHOCYTE PROLIFERATION;
MACROPHAGE ACTIVATION;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN DEFICIENCY;
PROTEIN EXPRESSION;
RECEPTOR UPREGULATION;
SKIN DISEASE;
SKIN LUPUS ERYTHEMATOSUS;
STRAIN DIFFERENCE;
SYSTEMIC DISEASE;
TARGET CELL;
|
EID: 0035451795
PISSN: 00221767
EISSN: None
Source Type: Journal
DOI: 10.4049/jimmunol.167.5.2985 Document Type: Article |
Times cited : (71)
|
References (74)
|