메뉴 건너뛰기




Volumn 167, Issue 5, 2001, Pages 2985-2990

Deficiency in β2-microglobulin, but not CD1, accelerates spontaneous lupus skin disease while inhibiting nephritis in MRL-Faslpr mice: An example of disease regulation at the organ level

Author keywords

[No Author keywords available]

Indexed keywords

BETA 2 MICROGLOBULIN; FAS ANTIBODY; IMMUNOGLOBULIN G; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 1;

EID: 0035451795     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.167.5.2985     Document Type: Article
Times cited : (71)

References (74)
  • 52
    • 0031985139 scopus 로고    scopus 로고
    • Suppression of immune responses by CD8 cells. I. Superantigen-activated CD8 cells induce unidirectional Fasmediated apoptosis of antigen-activated CD4 cells
    • (1998) J. Immunol. , vol.159 , pp. 559
    • Noble, A.1    Pestano, G.A.2    Cantor, H.3
  • 59
    • 0026056142 scopus 로고
    • IFN-γ modulates the early development of Th1 andTh2 responses in a murine model of cutaneous leishmaniasis
    • (1991) J. Immunol. , vol.147 , pp. 3149
    • Scott, P.1
  • 71
    • 0029994333 scopus 로고    scopus 로고
    • Systemic lupus erythematosus
    • (1996) Cell , vol.85 , pp. 303
    • Kotzin, B.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.