Oxidative damage to mitochondria is a preliminary step to caspase-3 activation in fluoride-induced apoptosis in HL-60 cells

Free Radical Biology and Medicine

Volumn 31, Issue 3, 2001, Pages 367-373

  Anuradha, Cunnigaipur Dhanasekaran ^a   Kanno, Sanae ^a   Hirano, Seishiro ^a  

^a NATIONAL INSTITUTE FOR ENVIRONMENTAL STUDIES   (Japan)

Author keywords

Apoptosis; Bcl 2; Cytochrome c; Fluoride; Free radicals; Lipid peroxides; Oxidative stress

Indexed keywords

4 HYDROXYNONENAL; ACETYLCYSTEINE; ANTIOXIDANT; BENZYLOXYCARBONYLVALYLALANYLASPARTYL FLUOROMETHYL KETONE; CASPASE 3; CASPASE INHIBITOR; CYTOCHROME C; FLUORIDE SODIUM; GLUTATHIONE; MALONALDEHYDE; PROTEIN BCL 2;

ANTIOXIDANT ACTIVITY; APOPTOSIS; ARTICLE; CELL PROTECTION; CELL STRAIN HL 60; CONTROLLED STUDY; CYTOSOL; DOWN REGULATION; ENZYME ACTIVATION; HUMAN; HUMAN CELL; LIPID PEROXIDATION; MEMBRANE POTENTIAL; MITOCHONDRION; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN SECRETION;

ACETYLCYSTEINE; ALDEHYDES; AMINO ACID CHLOROMETHYL KETONES; ANTIOXIDANTS; APOPTOSIS; CASPASE 3; CASPASES; CYSTEINE PROTEINASE INHIBITORS; GLUTATHIONE; HL-60 CELLS; HUMANS; LIPID PEROXIDATION; LIPID PEROXIDES; MALONDIALDEHYDE; MITOCHONDRIA; OXIDATIVE STRESS; SODIUM FLUORIDE;

EID: 0035426001     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0891-5849(01)00591-3     Document Type: Article

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