Overexpression of aromatase leads to hyperplasia and changes in the expression of genes involved in apoptosis, cell cycle, growth, and tumor suppressor functions in the mammary glands of transgenic mice

Cancer Research

Volumn 61, Issue 5, 2001, Pages 1910-1918

  Kirma, N ^a   Gill, K ^a   Mandava, U ^a   Tekmal, R R ^a  

^a EMORY UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AROMATASE; EPIDERMAL GROWTH FACTOR; EPIDERMAL GROWTH FACTOR RECEPTOR; ESTROGEN RECEPTOR; PROGESTERONE RECEPTOR; PROTEIN P53; RETINOBLASTOMA PROTEIN; TRANSFORMING GROWTH FACTOR ALPHA;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BREAST CARCINOGENESIS; BREAST HYPERPLASIA; CANCER INHIBITION; CELL CYCLE; CELL GROWTH; CONTROLLED STUDY; GENE EXPRESSION; MAMMARY GLAND; MOUSE; NONHUMAN; PRIORITY JOURNAL; TRANSGENIC MOUSE; TUMOR SUPPRESSOR GENE;

ANIMALS; APOPTOSIS; AROMATASE; BCL-2-ASSOCIATED X PROTEIN; BCL-X PROTEIN; CELL CYCLE; CELL DIVISION; ESTRADIOL; FEMALE; GENE EXPRESSION REGULATION; GENES, TUMOR SUPPRESSOR; GROWTH SUBSTANCES; HYPERPLASIA; MAMMARY GLANDS, ANIMAL; MAMMARY NEOPLASMS, EXPERIMENTAL; MICE; MICE, TRANSGENIC; PRECANCEROUS CONDITIONS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; RECEPTORS, ESTROGEN; RECEPTORS, PROGESTERONE;

EID: 0035266182     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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