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Volumn 113, Issue 1, 2001, Pages 109-118
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Lymphotoxin-alpha deficiency completely protects C57BL/6 mice from developing clinical experimental autoimmune myasthenia gravis
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Author keywords
Acetylcholine receptors; Autoimmunity; Cytokines; Lymphotoxins; Myasthenia gravis; TNF receptors
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Indexed keywords
CHOLINERGIC RECEPTOR;
CHOLINERGIC RECEPTOR ANTIBODY;
CYTOKINE;
IMMUNOGLOBULIN CLASS;
IMMUNOGLOBULIN G;
IMMUNOGLOBULIN G2A;
IMMUNOGLOBULIN G2B;
IMMUNOGLOBULIN M;
LYMPHOTOXIN;
TUMOR NECROSIS FACTOR RECEPTOR;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANTIBODY TITER;
ARTICLE;
AUTOIMMUNITY;
CONTROLLED STUDY;
CYTOKINE PRODUCTION;
FEMALE;
HUMORAL IMMUNITY;
IMMUNIZATION;
IMMUNOGLOBULIN GENE;
MALE;
MOUSE;
MYASTHENIA GRAVIS;
NONHUMAN;
PATHOGENESIS;
PRIORITY JOURNAL;
ANIMALS;
ANTIGENS, CD;
ANTIGENS, CD86;
AUTOANTIBODIES;
GENE EXPRESSION;
IMMUNODOMINANT EPITOPES;
IMMUNOGLOBULIN G;
IMMUNOGLOBULIN M;
LYMPHOTOXIN-ALPHA;
MEMBRANE GLYCOPROTEINS;
MICE;
MICE, INBRED C57BL;
MICE, KNOCKOUT;
MYASTHENIA GRAVIS, AUTOIMMUNE, EXPERIMENTAL;
RECEPTORS, CHOLINERGIC;
RECEPTORS, TUMOR NECROSIS FACTOR;
SPLEEN;
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EID: 0035255040
PISSN: 01655728
EISSN: None
Source Type: Journal
DOI: 10.1016/S0165-5728(00)00420-3 Document Type: Article |
Times cited : (22)
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References (36)
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