Transgenic mice overexpressing human KvLQT1 dominant-negative isoform. Part I: Phenotypic characterisation

Cardiovascular Research

Volumn 50, Issue 2, 2001, Pages 314-327

  Demolombe, Sophie ^a   Lande, Gilles ^a   Charpentier, Flavien ^a   Van Roon, Marian A ^b   Van Den Hoff, Maurice J B ^b   Toumaniantz, Gilles ^a   Baro, Isabelle ^a   Guihard, Gilles ^a   Le Berre, Nathalie ^c   Corbier, Alain ^c   De Bakker, Jacques ^b   Opthof, Tobias ^b   Wilde, Arthur ^b   Moorman, Antoon F M ^b   Escande, Denis ^a  

^a INSERM   (France)

^b ACADEMIC MEDICAL CENTER   (Netherlands)

^c HMR   (France)

Author keywords

Antiarrhythmic agents; Congenital defects; ECG; K channel; Long QT syndrome; Repolarisation

Indexed keywords

POTASSIUM CHANNEL; RIBONUCLEASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; ATRIOVENTRICULAR BLOCK; CONTROLLED STUDY; CORRELATION FUNCTION; FEMALE; GENE DISRUPTION; GENE OVEREXPRESSION; HEART ATRIOVENTRICULAR NODE; HEART MUSCLE POTENTIAL; HEART REPOLARIZATION; HUMAN; LONG QT SYNDROME; MALE; MOLECULAR CLONING; MOUSE; NONHUMAN; PATCH CLAMP; PHENOTYPE; POTASSIUM CURRENT; PRIORITY JOURNAL; SINUS NODE; TRANSGENIC MOUSE; WENCKEBACH PERIOD;

EID: 0035038277     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0008-6363(01)00231-0     Document Type: Article

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