Attenuation of the slow component of delayed rectification, action potential prolongation, and triggered activity in mice expressing a dominant- negative Kv2 α subunit

Circulation Research

Volumn 85, Issue 7, 1999, Pages 623-633

  Xu, Haodong ^a   Barry, Dianne M ^a   Li, Huilin ^a   Brunet, Sylvain ^a   Guo, Weinong ^a   Nerbonne, Jeanne M ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Action potential; Arrhythmia; Transgenic mice; Triggered activity; Ventricle

Indexed keywords

4 AMINOPYRIDINE; POTASSIUM CHANNEL; POTASSIUM ION;

ACTION POTENTIAL; ALPHA CHAIN; AMINO TERMINAL SEQUENCE; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CARBOXY TERMINAL SEQUENCE; CHANNEL GATING; DEPOLARIZATION; HEART VENTRICLE FUNCTION; ION CURRENT; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN EXPRESSION; QT INTERVAL; TRANSGENIC MOUSE;

EID: 0033215099     PISSN: 00097330     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.RES.85.7.623     Document Type: Article

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