Peroxisome proliferator-activated receptor-γ expression in human malignant and normal brain, breast and prostate-derived cells

Prostaglandins Leukotrienes and Essential Fatty Acids

Volumn 64, Issue 4-5, 2001, Pages 241-245

  Nwankwo, J O ^a   Robbins, M E C ^a  

^a UNIVERSITY OF IOWA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GAMMA LINOLENIC ACID; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA;

ARTICLE; ASTROCYTOMA; BRAIN CELL; BREAST CARCINOMA; BREAST CELL; CONTROLLED STUDY; GLIOBLASTOMA; HUMAN; HUMAN CELL; IMMUNOBLOTTING; PRIORITY JOURNAL; PROSTATE; PROSTATE CARCINOMA; PROTEIN EXPRESSION;

EID: 0034943192     PISSN: 09523278     EISSN: None     Source Type: Journal    
DOI: 10.1054/plef.2001.0266     Document Type: Article

References (31)

1. Identification of a peroxisome proliferator-responsive element upstream of the gene encoding rat peroxisomal enoyl-CoA hydratase 3-hydroxyacyl-CoA dehydrogenase
2. Hypolipidemic drugs, polyunsaturated fatty acids, and eicosanoids are ligands for peroxisome proliferator-activated receptors α and δ
3. Fatty acids, eicosanoids, and hypolipidemic agents identified as ligands of peroxisome proliferator-activated receptors by coactivator-dependent receptor ligand assay
4. Fatty acids and eicosanoids regulate gene expression through direct interactions with peroxisome proliferator-activated receptors alpha and gamma
5. The nuclear receptor superfamily: The second decade
6. Differential expression and activation of a family of murine peroxisomal proliferator-activated receptors
7. 4 pathway to inflammation control
8. Activation of human aortic smooth-muscle cells is inhibited by PPARalpha but not be PPARgama activators
9. mPPARγ2: Tissue-specific regulator of an adipocyte enhancer
10. Organization, promoter analysis and expression of the human PPARγ gene
11. Tissue distribution and quantification of the expression of PPARs and LXRα in humans: No alterations in adipose tissue of obese and NIDDM patients
12. Terminal differentiation of human breast cancer through PPARγ
13. Differentiation and reversal of malignant changes in colon cancer through PPAR gamma
14. Activation of PPARγ inhibits cell growth and induces apoptosis in human gastric cancer cells
15. Inhibition of human lung cancer cell growth by the peroxisome proliferator-activated receptor-γ agonists through induction of apoptosis
16. 2
17. Molecular recognition of fatty acids by peroxisome proliferator-activated receptors
18. Polyunsaturated fatty acids augment free radical generation in tumor cells in vitro
19. γ-Linolenic acid (GLA) is cytotoxic to 36B10 malignant rat astrocytoma cells but not to normal rat astrocytes
20. Superoxide dismutase: Improved assays and an assay applicable to acrylamide gels
21. Peroxisome proliferator-activated receptor gamma is induced during differentiation of colon epithelium cells
22. Effects of ligand activation of peroxisome proliferator-activated receptor in human prostate cancer
23. Induction of differentiation and apoptosis by ligands of peroxisome proliferator-activated receptor γ in non-small cell lung cancer
24. Expression of peroxisome proliferator-activated receptors (PPARs) in human astrocytic cells: PPARγ agonists as inducers of apoptosis
25. Medical significance of peroxisome proliferator-activated receptors
26. Troglitazone increases the number of small adipocytes without the change of white adipose tissue mass in obese zucker rats
27. Ligand for peroxisome proliferator-activated receptor γ (Troglitazone) has potent antitumor effect against human prostate cancer both in vitro and in vivo
28. Ligands for peroxisome proliferator-activated receptor γ and retinoic acid receptor inhibit growth and induce apoptosis of human breast cancer cell in vitro and in BNX mice
29. Loss-of-function mutations in PPARγ associated with human colon cancer
30. c-Jun n-terminal kinase phosphorylates peroxisome proliferator-activated receptor-γ1 and negatively regulates its transcriptional activity
31. Transcriptional activation by peroxisome proliferator-activated receptor γ is inhibited by phosphorylation at a consensus mitogen-activated protein kinase site