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Volumn 12, Issue 3, 2001, Pages 137-145
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Activation of c-Jun NH2-terminal kinase/stress-activated protein kinase (JNK/SAPK) is critical for hypoxia-induced apoptosis of human malignant melanoma
a
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Author keywords
[No Author keywords available]
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Indexed keywords
2 (2 AMINO 3 METHOXYPHENYL)CHROMONE;
4 (4 FLUOROPHENYL) 2 (4 METHYLSULFINYLPHENYL) 5 (4 PYRIDYL)IMIDAZOLE;
FAS LIGAND;
MUTANT PROTEIN;
STRESS ACTIVATED PROTEIN KINASE;
SYNAPTOPHYSIN;
AMINO TERMINAL SEQUENCE;
APOPTOSIS;
ARTICLE;
CONTROLLED STUDY;
CORRELATION FUNCTION;
ENZYME ACTIVATION;
ENZYME INHIBITION;
GENE OVEREXPRESSION;
HUMAN;
HUMAN CELL;
HYPOXIA;
IMMUNOHISTOCHEMISTRY;
MELANOMA CELL;
METASTASIS;
PRIORITY JOURNAL;
REGULATORY MECHANISM;
SIGNAL TRANSDUCTION;
TUMOR CELL;
ANOXIA;
ANTIGENS, CD95;
APOPTOSIS;
GENE EXPRESSION REGULATION, NEOPLASTIC;
HUMANS;
IMMUNOHISTOCHEMISTRY;
JNK MITOGEN-ACTIVATED PROTEIN KINASES;
MAP KINASE KINASE 4;
MELANOMA;
MITOGEN-ACTIVATED PROTEIN KINASE KINASES;
MITOGEN-ACTIVATED PROTEIN KINASES;
NECROSIS;
NEOPLASM INVASIVENESS;
NEOVASCULARIZATION, PATHOLOGIC;
PROTO-ONCOGENE PROTEINS C-JUN;
RNA, MESSENGER;
TRANSCRIPTION, GENETIC;
TRANSFECTION;
TUMOR CELLS, CULTURED;
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EID: 0034920088
PISSN: 10449523
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
Times cited : (57)
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References (46)
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