Mitochondrial KATP channel opening protects a human atrial-derived cell line by a mechanism involving free radical generation

Cardiovascular Research

Volumn 51, Issue 4, 2001, Pages 691-700

  Carroll, Richard ^a   Gant, Vanya A ^a   Yellon, Derek M ^a  

^a ROYAL FREE AND UNIVERSITY COLLEGE MEDICAL SCHOOL   (United Kingdom)

Author keywords

Free radicals; K ATP channel; Mitochondria; Preconditioning

Indexed keywords

5 HYDROXYDECANOIC ACID; DIAZOXIDE; POTASSIUM CHANNEL; PROPIDIUM IODIDE; REACTIVE OXYGEN METABOLITE; TIOPRONIN;

ARTICLE; CELL LINE; CELL SURVIVAL; CHANNEL GATING; CONTROLLED STUDY; DISEASE MODEL; DRUG EFFECT; DRUG MECHANISM; HEART ATRIUM; HEART MITOCHONDRION; HEART MUSCLE CELL; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PROTECTION; HUMAN; HUMAN CELL; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; MITOCHONDRION SWELLING; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ANALYSIS OF VARIANCE; ANTI-ARRHYTHMIA AGENTS; CELL LINE; DECANOIC ACIDS; DIAZOXIDE; FLOW CYTOMETRY; FREE RADICAL SCAVENGERS; HEART ATRIA; HUMANS; HYDROXY ACIDS; ION CHANNEL GATING; ISCHEMIC PRECONDITIONING, MYOCARDIAL; MEMBRANE POTENTIALS; MICROSCOPY, FLUORESCENCE; MITOCHONDRIA, HEART; POTASSIUM CHANNELS; REACTIVE OXYGEN SPECIES; THIOPRONINE;

EID: 0034860570     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0008-6363(01)00330-3     Document Type: Article

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