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Volumn 19, Issue 48, 2000, Pages 5444-5452

The integrin linked kinase (ILK) induces an invasive phenotype via AP-1 transcription factor-dependent upregulation of matrix metalloproteinase 9 (MMP-9)

(6) Troussard, Armelle A a Costello, Penny b Yoganathan, T Nathan b Kumagai, Shigehiro c Roskelley, Calvin D d Dedhar, Shoukat a,d

a BRITISH COLUMBIA CANCER AGENCY (Canada)

b Kinetek Pharmaceuticals Inc (Canada)

c KANAZAWA UNIVERSITY (Japan)

d UNIVERSITY OF BRITISH COLUMBIA (Canada)

Author keywords

AP 1 transcription factor; Integrins; Matrix metalloproteinases; Signal transduction; Tumor invasion

Indexed keywords

GELATINASE B; GLYCOGEN SYNTHASE KINASE 3; INTEGRIN; INTEGRIN LINKED KINASE; PROTEIN KINASE; TRANSCRIPTION FACTOR AP 1; UNCLASSIFIED DRUG;

ANIMAL CELL; ARTICLE; BRAIN TUMOR; CANCER INVASION; CONTROLLED STUDY; ENZYME ACTIVITY; HUMAN; HUMAN CELL; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN INDUCTION; RAT; SIGNAL TRANSDUCTION; TUMOR CELL LINE;

ANIMALS; BINDING SITES; CELL LINE; GENE EXPRESSION REGULATION, ENZYMOLOGIC; GLIOBLASTOMA; HUMANS; MATRIX METALLOPROTEINASE 9; MICE; PHENOTYPE; PROMOTER REGIONS (GENETICS); PROTEIN-SERINE-THREONINE KINASES; TRANSCRIPTION FACTOR AP-1; TRANSFECTION; UP-REGULATION;

EID: 0034676338 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1203928 Document Type: Article

Times cited : (171)

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