Glucocorticoids antagonize AP-1 by inhibiting the activation/phosphorylation of JNK without affecting its subcellular distribution

Journal of Cell Biology

Volumn 150, Issue 5, 2000, Pages 1199-1207

  González, María Victoria ^a   Jiménez, Benilde ^a   Berciano, María T ^b   González Sancho, José Manuel ^a   Caelles, Carme ^c   Lafarga, Miguel ^b   Muñoz, Alberto ^a  

^a UNIVERSIDAD AUTÓNOMA DE MADRID   (Spain)

^b UNIVERSITY OF CANTABRIA   (Spain)

^c UNIVERSITY OF BARCELONA   (Spain)

Author keywords

Activating protein 1; C Jun NH2 terminal kinase; Dexamethasone; Nuclear translocation; Tumor necrosis factor

Indexed keywords

DEXAMETHASONE; GLUCOCORTICOID; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; STRESS ACTIVATED PROTEIN KINASE; TRANSCRIPTION FACTOR AP 1; TUMOR NECROSIS FACTOR;

ANIMAL CELL; ARTICLE; CELL NUCLEUS; CELLULAR DISTRIBUTION; CONTROLLED STUDY; CYTOPLASM; DIMERIZATION; ENZYME ACTIVATION; ENZYME ANALYSIS; ENZYME PHOSPHORYLATION; HUMAN; HUMAN CELL; NONHUMAN; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ANIMALS; CELL FRACTIONATION; CELL NUCLEUS; COS CELLS; CYTOSOL; DEXAMETHASONE; ENZYME ACTIVATION; GLUCOCORTICOIDS; HELA CELLS; HUMANS; JNK MITOGEN-ACTIVATED PROTEIN KINASES; KINETICS; MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; RECOMBINANT PROTEINS; SIGNAL TRANSDUCTION; TRANSCRIPTION FACTOR AP-1; TRANSFECTION; TUMOR NECROSIS FACTOR-ALPHA;

ANIMALIA;

EID: 0034605125     PISSN: 00219525     EISSN: None     Source Type: Journal    
DOI: 10.1083/jcb.150.5.1199     Document Type: Article

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