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Biochemical and Biophysical Research Communications

Volumn 274, Issue 1, 2000, Pages 16-21

Processing of cdk5 activator p35 to its truncated form (p25) by calpain in acutely injured neuronal cells

(7) Nath, Rathna a Davis, Megan a Probert, Albert W a Kupina, Nancy C a Ren, Xiaodong a Schielke, Gerald P a Wang, Kevin K W a,b

a PFIZER INC (United States)

b UNIVERSITY OF MICHIGAN MEDICAL SCHOOL (United States)

Author keywords

Alzheimer's disease; Calpain; cdk5; Neurodegeneration; p25

Indexed keywords

ANTIBODY; CALPAIN; CALPASTATIN; CASPASE INHIBITOR; FODRIN; LACTACYSTIN; MAITOTOXIN; PROTEASOME INHIBITOR; PROTEIN P35; TAU PROTEIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BRAIN ISCHEMIA; CEREBELLUM; CONTROLLED STUDY; ENZYME ACTIVITY; GRANULE CELL; MALE; MIDDLE CEREBRAL ARTERY OCCLUSION; NERVE CELL LESION; NONHUMAN; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; PROTEIN PROCESSING; RAT;

EID: 0033915457 PISSN: 0006291X EISSN: None Source Type: Journal
DOI: 10.1006/bbrc.2000.3070 Document Type: Article

Times cited : (152)

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