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Volumn 52, Issue 4, 2000, Pages 369-379
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Hypersusceptibility of A/J mice to tuberculosis is in part due to a deficiency of the fifth complement component (C5)
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Author keywords
[No Author keywords available]
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Indexed keywords
CHEMOKINE;
COMPLEMENT COMPONENT C5;
INTERLEUKIN 1BETA;
INTERLEUKIN 6;
TUMOR NECROSIS FACTOR ALPHA;
AEROSOL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
CELL KILLING;
CHRONIC DISEASE;
CONTROLLED STUDY;
CYTOKINE RELEASE;
DISEASE EXACERBATION;
INFECTION SENSITIVITY;
MACROPHAGE ACTIVATION;
MOUSE;
MYCOBACTERIUM TUBERCULOSIS;
NONHUMAN;
PRIORITY JOURNAL;
TUBERCULOSIS;
ANIMALS;
CHEMOKINES;
COMPLEMENT C5;
CYTOKINES;
DISEASE SUSCEPTIBILITY;
FEMALE;
HYDROGEN PEROXIDE;
MACROPHAGE ACTIVATION;
MACROPHAGES;
MICE;
MICE, INBRED C57BL;
MYCOBACTERIUM TUBERCULOSIS;
NITRIC OXIDE;
TUBERCULOSIS;
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EID: 0033834379
PISSN: 03009475
EISSN: None
Source Type: Journal
DOI: 10.1046/j.1365-3083.2000.00770.x Document Type: Article |
Times cited : (53)
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References (49)
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