P53 knockout mice (-/-) are more susceptible than (+/-) or (+/+) mice to N-methyl-N-nitrosourea stomach carcinogenesis

Carcinogenesis

Volumn 21, Issue 10, 2000, Pages 1891-1897

  Yamamoto, Masami ^a   Tsukamoto, Tetsuya ^a   Sakai, Hiroki ^a   Shirai, Norimitsu ^a   Ohgaki, Hiroko ^b   Furihata, Chie ^c   Donehower, Lawrence A ^d   Yoshida, Kenji ^e   Tatematsu, Masae ^a  

^a AICHI CANCER CENTER HOSPITAL   (Japan)

^b INTERNATIONAL AGENCY FOR RESEARCH ON CANCER   (France)

^c AOYAMA GAKUIN UNIVERSITY   (Japan)

^d BAYLOR COLLEGE OF MEDICINE   (United States)

^e AICHI GAKUIN UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

METHYLNITROSOUREA; PEPSINOGEN; PROTEIN P53; WATER;

ADENOMA; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CANCER MORTALITY; CANCER SUSCEPTIBILITY; CONTROLLED STUDY; DNA POLYMORPHISM; FEMALE; GENE MUTATION; GENETIC SUSCEPTIBILITY; GENOTOXICITY; HETEROZYGOSITY LOSS; INCIDENCE; KNOCKOUT MOUSE; LEUKEMIA; MALE; MOLECULAR INTERACTION; MOUSE; NONHUMAN; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; PROTEIN DEFICIENCY; PYLORUS; STOMACH ADENOCARCINOMA; STOMACH CARCINOGENESIS; STOMACH GLAND; STOMACH LYMPHOMA; STOMACH SARCOMA; STOMACH TUMOR; TUMOR SUPPRESSOR GENE;

EID: 0033790792     PISSN: 01433334     EISSN: None     Source Type: Journal    
DOI: 10.1093/carcin/21.10.1891     Document Type: Article

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