Reactive astrocytes upregulate Fas (CD95) and Fas ligand (CD95L) expression but do not undergo programmed cell death during the course of anterograde degeneration

GLIA

Volumn 32, Issue 1, 2000, Pages 25-41

  Bechmann, Ingo ^a   Lossau, Sandra ^a   Steiner, Barbara ^a   Mor, Gil ^b   Gimsa, Ulrike ^a   Nitsch, Robert ^a  

^a CHARITÉ UNIVERSITÄTSMEDIZIN BERLIN   (Germany)

^b YALE UNIVERSITY   (United States)

Author keywords

Anterograde degeneration; Astrocyte upregulation; Fas; FasL

Indexed keywords

APOPTOSIS SPECIFIC PROTEIN; CYTOSKELETON PROTEIN; DIAGNOSTIC AGENT; FAS ANTIGEN; FASL PROTEIN; GLIAL FIBRILLARY ACIDIC PROTEIN; HEMATOXYLIN; MEMBRANE PROTEIN; PROTEIN C JUN; PROTO ONCOGENE PROTEINS C JUN;

ANIMAL; APOPTOSIS; ARTICLE; ASTROCYTE; CELL NUCLEUS; DENERVATION; ENTORHINAL CORTEX; METABOLISM; NERVE DEGENERATION; NICK END LABELING; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; RAT; RAT STRAIN; ULTRASTRUCTURE;

ANIMAL; ANTIGENS, CD95; APOPTOSIS; ASTROCYTES; CELL NUCLEUS; CYTOSKELETAL PROTEINS; DENERVATION; ENTORHINAL CORTEX; GLIAL FIBRILLARY ACIDIC PROTEIN; HEMATOXYLIN; IN SITU NICK-END LABELING; MEMBRANE GLYCOPROTEINS; PROTO-ONCOGENE PROTEINS C-JUN; RATS; RATS, WISTAR; SUPPORT, NON-U.S. GOV'T; WALLERIAN DEGENERATION;

EID: 0033771698     PISSN: 08941491     EISSN: None     Source Type: Journal    
DOI: 10.1002/1098-1136(200010)32:1<25::AID-GLIA30>3.0.CO;2-Y     Document Type: Article

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