The common PPARγ Pro12Ala polymorphism is associated with decreased risk of type 2 diabetes

Nature Genetics

Volumn 26, Issue 1, 2000, Pages 76-80

  Altshuler, David ^a,b,c   Hirschhorn, Joel N ^a,c,d   Klannemark, Mia ^e   Lindgren, Cecilia M ^a,e   Vohl, Marie Claude ^f   Nemesh, James ^a   Lane, Charles R ^a   Schaffner, Stephen F ^a   Bolk, Stacey ^a   Brewer, Carl ^f   Tuomi, Tiinamaija ^e,g   Gaudet, Daniel ^h   Hudson, Thomas J ^a,f   Daly, Mark ^a   Groop, Leif ^e   Lander, Eric S ^a,i  

^a WHITEHEAD INSTITUTE FOR BIOMEDICAL RESEARCH   (United States)

^b MASSACHUSETTS GENERAL HOSPITAL   (United States)

^c HARVARD MEDICAL SCHOOL   (United States)

^d BOSTON CHILDREN S HOSPITAL   (United States)

^e LUND UNIVERSITY HOSPITAL   (Sweden)

^f RESEARCH INSTITUTE OF THE MCGILL UNIVERSITY HEALTH CENTRE   (Canada)

^g HELSINKI UNIVERSITY CENTRAL HOSPITAL   (Finland)

^h CHICOUTIMI HOSPITAL   (Canada)

ⁱ MASSACHUSETTS INSTITUTE OF TECHNOLOGY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR; ALANINE; CELL RECEPTOR; CHOLESTEROL; HIGH DENSITY LIPOPROTEIN; PROLINE; TRANSCRIPTION FACTOR;

ADULT; ARTICLE; CONTROLLED STUDY; FEMALE; GENETIC ANALYSIS; GENETIC ASSOCIATION; GENETIC POLYMORPHISM; GENOTYPE; HUMAN; MAJOR CLINICAL STUDY; MALE; NON INSULIN DEPENDENT DIABETES MELLITUS; PATHOGENESIS; PHENOTYPE; PRIORITY JOURNAL; AGED; ALLELE; BIOLOGICAL MODEL; BLOOD PRESSURE; BODY MASS; CLINICAL TRIAL; FAMILY HEALTH; FATHER; GENE LINKAGE DISEQUILIBRIUM; GENETICS; GLUCOSE BLOOD LEVEL; META ANALYSIS; MIDDLE AGED; MOTHER; ONSET AGE; RISK FACTOR;

ADULT; AGE OF ONSET; AGED; ALANINE; ALLELES; BLOOD GLUCOSE; BLOOD PRESSURE; BODY MASS INDEX; CHOLESTEROL; DIABETES MELLITUS, TYPE 2; FAMILY HEALTH; FATHERS; FEMALE; GENOTYPE; HUMANS; LINKAGE DISEQUILIBRIUM; LIPOPROTEINS, HDL; MALE; MIDDLE AGED; MODELS, GENETIC; MOTHERS; PHENOTYPE; POLYMORPHISM, GENETIC; PROLINE; RECEPTORS, CYTOPLASMIC AND NUCLEAR; RISK FACTORS; TRANSCRIPTION FACTORS;

EID: 0033624575     PISSN: 10614036     EISSN: None     Source Type: Journal    
DOI: 10.1038/79216     Document Type: Article

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