MRL-lpr/lpr mice exhibit a defect in maintaining developmental arrest and follicular exclusion of anti-double-stranded DNA B cells

Journal of Experimental Medicine

Volumn 189, Issue 11, 1999, Pages 1799-1814

  Mandik Nayak, Laura ^a   Seo, Su Jean ^a   Sokol, Caroline ^a   Potts, Kathryn M ^a   Bui, Anh ^a   Erikson, Jan ^a  

^a WISTAR INSTITUTE   (United States)

Author keywords

Antinuclear antibody; Autoimmunity; Fas; Splenic architecture; Tolerance

Indexed keywords

DOUBLE STRANDED DNA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANTIBODY PRODUCTION; ARTICLE; AUTOIMMUNITY; B LYMPHOCYTE; IMMUNOLOGICAL TOLERANCE; LYMPH FOLLICLE; MOUSE; NONHUMAN; PRIORITY JOURNAL; SYSTEMIC LUPUS ERYTHEMATOSUS;

ANIMALS; ANTIBODIES, ANTINUCLEAR; ANTIGENS, CD; ANTIGENS, CD95; AUTOIMMUNITY; B-LYMPHOCYTES; CD4-POSITIVE T-LYMPHOCYTES; CELL DIFFERENTIATION; LUPUS ERYTHEMATOSUS, SYSTEMIC; MICE; MICE, INBRED BALB C; MICE, INBRED MRL LPR; MICE, TRANSGENIC;

EID: 0033532630     PISSN: 00221007     EISSN: None     Source Type: Journal    
DOI: 10.1084/jem.189.11.1799     Document Type: Article

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